Genome-Wide Search for Gene-Gene Interactions in Colorectal Cancer

نویسندگان

  • Shuo Jiao
  • Li Hsu
  • Sonja Berndt
  • Stéphane Bézieau
  • Hermann Brenner
  • Daniel Buchanan
  • Bette J. Caan
  • Peter T. Campbell
  • Christopher S. Carlson
  • Graham Casey
  • Andrew T. Chan
  • Jenny Chang-Claude
  • Stephen Chanock
  • David V. Conti
  • Keith R. Curtis
  • David Duggan
  • Steven Gallinger
  • Stephen B. Gruber
  • Tabitha A. Harrison
  • Richard B. Hayes
  • Brian E. Henderson
  • Michael Hoffmeister
  • John L. Hopper
  • Thomas J. Hudson
  • Carolyn M. Hutter
  • Rebecca D. Jackson
  • Mark A. Jenkins
  • Elizabeth D. Kantor
  • Laurence N. Kolonel
  • Sébastien Küry
  • Loic Le Marchand
  • Mathieu Lemire
  • Polly A. Newcomb
  • John D. Potter
  • Conghui Qu
  • Stephanie A. Rosse
  • Robert E. Schoen
  • Fred R. Schumacher
  • Daniela Seminara
  • Martha L. Slattery
  • Cornelia M. Ulrich
  • Brent W. Zanke
  • Ulrike Peters
چکیده

Genome-wide association studies (GWAS) have successfully identified a number of single-nucleotide polymorphisms (SNPs) associated with colorectal cancer (CRC) risk. However, these susceptibility loci known today explain only a small fraction of the genetic risk. Gene-gene interaction (GxG) is considered to be one source of the missing heritability. To address this, we performed a genome-wide search for pair-wise GxG associated with CRC risk using 8,380 cases and 10,558 controls in the discovery phase and 2,527 cases and 2,658 controls in the replication phase. We developed a simple, but powerful method for testing interaction, which we term the Average Risk Due to Interaction (ARDI). With this method, we conducted a genome-wide search to identify SNPs showing evidence for GxG with previously identified CRC susceptibility loci from 14 independent regions. We also conducted a genome-wide search for GxG using the marginal association screening and examining interaction among SNPs that pass the screening threshold (p<10(-4)). For the known locus rs10795668 (10p14), we found an interacting SNP rs367615 (5q21) with replication p = 0.01 and combined p = 4.19×10(-8). Among the top marginal SNPs after LD pruning (n = 163), we identified an interaction between rs1571218 (20p12.3) and rs10879357 (12q21.1) (nominal combined p = 2.51×10(-6); Bonferroni adjusted p = 0.03). Our study represents the first comprehensive search for GxG in CRC, and our results may provide new insight into the genetic etiology of CRC.

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عنوان ژورنال:

دوره 7  شماره 

صفحات  -

تاریخ انتشار 2012